Download Clinical Pain Management: A Practical Guide by Mary E. Lynch, Kenneth D. Craig, Philip W. H. Peng PDF

By Mary E. Lynch, Kenneth D. Craig, Philip W. H. Peng

Scientific soreness administration takes a realistic, interdisciplinary method of the overview and administration of ache. Concise template chapters function a brief connection with physicians, anesthetists and neurologists, in addition to different experts, generalists, and trainees handling discomfort. in keeping with the overseas organization for the examine of Pain’s medical curriculum at the subject, this reference offers to-the-point best-practice tips in an easy-to-follow structure together with tables, bullets, algorithms and instructions.

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Additional resources for Clinical Pain Management: A Practical Guide

Sample text

Can Psychol 50(1):22–32. 6 Mogil JS. (2009) Are we getting anywhere in human pain genetics? Pain 146(3):231–2. 7 Mailis-Gagnon A, Yegneswaran B, Lakha SF et al. (2007) Pain characteristics and demographics of patients attending a university-affiliated pain clinic in Toronto, Ontario. Pain Res Manag 12(2):93–9. 8 Melzack R, Katz J. (2005) Pain assessment in adult patients. In: McMahon SB, Koltzenburg M, eds. Melzack and Wall’s Textbook of Pain, 5th edn. Churchill-Livingstone. pp. 291–304. 9 Carragee EJ, Don AS, Hurwitz EL et al.

It is featured in the widely endorsed definition of pain: “An unpleasant sensory and emotional experience associated with actual or potential tissue damage, or described in terms of such damage” [1]. The definition carefully explains that tissue damage is associated with or described by the person as a feature of the experience, but it establishes that it is not necessarily the exclusive or sufficient cause, thereby pointing to important roles for psychosocial determinants. Increasingly well-defined psychological factors should be considered when attempting to understand an individual’s unique pattern of pain experience and expression.

The peripheral nociceptive barrage associated with surgery, central sensitization) is a causal risk factor for CPSP. However, if the relationship between acute postoperative pain and CPSP is merely correlative, and both are caused by one or more factors that themselves are inter-related, then no type or amount of blocking will prevent the development of CPSP (Figure 1, bottom panel). The focus of preventive analgesia is on attenuating the impact of the peripheral nociceptive barrage associated with noxious preoperative, intraoperative and/or postoperative events and/or stimuli.

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