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Additional info for Clinical Toxicology - Vol 48, issue 7, Aug 2010

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Clinical Toxicology vol. 48 no. 7 2010 708 151. Ramamoorthy Y, Tyndale RF, Sellers EM. 10 differ in catalytic activity for multiple substrates. Pharmacogenetics 2001; 11:477–487. 152. Morgan D, Grant KA, Gage HD, et al. Social dominance in monkeys: dopamine D2 receptors and cocaine self-administration. Nat Neurosci 2002; 5:169–174. D. Seger 153. Kreth K, Kovar K, Schwab M, Zanger UM. Identification of the human cytochromes P450 involved in the oxidative metabolism of “Ecstasy”related designer drugs.

35. Reisch A, Illing R, Laszig R. Immediate early gene expression invoked by electrical intracochlear stimulation in some but not all types of neurons in the rat auditory brainstem. Exp Neurol 2007; 208:193–206. Clinical Toxicology vol. 48 no. 72010 Neuroadaptation 36. Kubik S, Miyashita T, Guzowski J. Using immediate-early genes to map hippocampal subregional functions. Learn Mem 2007; 14:758–770. 37. Thomas MJ, Kalivas PW, Shaham Y. Neuroplasticity in the mesolimbic dopamine system and cocaine addiction.

3 mmol/kg) (n = 5–6 per condition). No OHC or sodium nitrate animals survived at the lowest dose of cyanide gas. All cobinamide-treated animals survived, whereas 80%, 60%, and none of sodium thiosulfate-treated animals survived when treated with 534, 801, and 908 ppm, respectively. The combination of sodium thiosulfate and sodium nitrite was fully effective except when 908 ppm cyanide was administered, in which only 20% of animals survived (n = 5). The data were plotted and log transformed for nonlinear regression analysis.

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